Publications for All Faculty
The contribution of muscle to whole-body protein turnover throughout the course of burn injury in children.
av Integrin expression by DCs is required for Th17 cell differentiation and development of experimental autoimmune encephalomyelitis in mice.
Mining the plant-herbivore interface with a leafmining Drosophila of Arabidopsis.
Stem cells and burns: review and therapeutic implications.
Predicting success of oligomerized pool engineering (OPEN) for zinc finger target site sequences.
Chemoselective multicomponent one-pot assembly of purine precursors in water.
Ubiquitin accumulation in autophagy-deficient mice is dependent on the Nrf2-mediated stress response pathway: a potential role for protein aggregation in autophagic substrate selection.
LRRK2 is involved in the IFN-gamma response and host response to pathogens.
DNA ends: just the beginning (Nobel lecture).
Discover and connect cellular signaling.
Morphology-guided graph search for untangling objects: C. elegans analysis.
NLRP3 inflammasome plays a key role in the regulation of intestinal homeostasis.
Mutation of the ATP cassette binding transporter A1 (ABCA1) C-terminus disrupts HIV-1 Nef binding but does not block the Nef enhancement of ABCA1 protein degradation.
Plasma proteome response to severe burn injury revealed by 18O-labeled "universal" reference-based quantitative proteomics.
Effect of simvastatin on burn-induced alterations in tissue specific glucose metabolism: implications for burn associated insulin resistance.
Mobility of plasmids.
Clinical microfluidics for neutrophil genomics and proteomics.
Human IRGM gene "to be or not to be".
Autonomous zinc-finger nuclease pairs for targeted chromosomal deletion.
Engineering single Cys2His2 zinc finger domains using a bacterial cell-based two-hybrid selection system.
Single-molecule binding experiments on long time scales.
Circulating Nef induces dyslipidemia in simian immunodeficiency virus-infected macaques by suppressing cholesterol efflux.
Burn injury reduces neutrophil directional migration speed in microfluidic devices.
Reactive bone marrow stromal cells attenuate systemic inflammation via sTNFR1.
Genome-wide analysis reveals mechanisms modulating autophagy in normal brain aging and in Alzheimer's disease.