Publications for All Faculty
A High-content Imaging Workflow to Study Grb2 Signaling Complexes by Expression Cloning.
Prediction of C. elegans Longevity Genes by Human and Worm Longevity Networks.
Common alleles that influence autophagy and the risk for inflammatory bowel disease.
Imaging C-Fos Gene Expression in Burns Using Lipid Coated Spion Nanoparticles.
Engineering Designer Transcription Activator--Like Effector Nucleases (TALENs) by REAL or REAL-Fast Assembly.
The Two-Component Sensor KinB Acts as a Phosphatase to Regulate Pseudomonas aeruginosa Virulence.
Dysfunction of the intestinal microbiome in inflammatory bowel disease and treatment.
Bacille Calmette-Guerin induces NOD2-dependent nonspecific protection from reinfection via epigenetic reprogramming of monocytes.
Essential cell autonomous role for interferon regulatory factor 1 in interferon-?-mediated inhibition of norovirus replication in macrophages.
CDPKs in immune and stress signaling.
Evaluation of OPEN Zinc Finger Nucleases for Direct Gene Targeting of the ROSA26 Locus in Mouse Embryos.
Measuring the cost of care for children with acute burn injury.
Impact of hand burns on health-related quality of life in children younger than 5 years.
The American Burn Association/Shriners Hospitals for Children Burn Outcomes Program: A progress report at 15 years.
Previous burn injury predisposes mice to lipopolysaccharide-induced changes in glucose metabolism.
Safety and dosing of bevacizumab (avastin) for the treatment of recurrent respiratory papillomatosis.
Complexity in Differential Peptide-Receptor Signaling: Response to Segonzac et al. and Mueller et al. Commentaries.
FLS2-Mediated Responses to Ax21-Derived Peptides: Response to the Mueller et al. Commentary.
p40phox Expression Regulates Neutrophil Recruitment and Function during the Resolution Phase of Intestinal Inflammation.
Multicomponent assembly of proposed DNA precursors in water.
Enhanced Tim3 Activity Improves Survival after Influenza Infection.
Enhanced Tim3 activity improves survival after influenza infection.
Candida albicans Infection Affords Protection against Reinfection via Functional Reprogramming of Monocytes.
New insights into siRNA amplification and RNAi.
A synthetic biology framework for programming eukaryotic transcription functions.
