Publications for All Faculty
Differential Effects of Environmental and Genetic Factors on T and B Cell Immune Traits.
Unusual Base-Pairing Interactions in Monomer-Template Complexes.
Linking the Human Gut Microbiome to Inflammatory Cytokine Production Capacity.
Host and Environmental Factors Influencing Individual Human Cytokine Responses.
A Functional Genomics Approach to Understand Variation in Cytokine Production in Humans.
RNF166 Determines Recruitment of Adaptor Proteins during Antibacterial Autophagy.
Vedolizumab Therapy Is Associated with an Improvement in Sleep Quality and Mood in Inflammatory Bowel Diseases.
KERIS: kaleidoscope of gene responses to inflammation between species.
SIKs control osteocyte responses to parathyroid hormone.
Interplay of host genetics and gut microbiota underlying the onset and clinical presentation of inflammatory bowel disease.
TNF-?/IL-10 Ratio Correlates with Burn Severity and May Serve as a Risk Predictor of Increased Susceptibility to Infections.
Corrigendum: Inter-individual variability and genetic influences on cytokine responses to bacteria and fungi.
The metabolic stress response to burn trauma: current understanding and therapies.
A perspective on single cell behavior during infection.
Succinate Dehydrogenase Supports Metabolic Repurposing of Mitochondria to Drive Inflammatory Macrophages.
Changes in neutrophil migration patterns upon contact with platelets in a microfluidic assay.
Erratum: A protein-truncating R179X variant in RNF186 confers protection against ulcerative colitis.
Increased Intestinal Microbial Diversity Following Fecal Microbiota Transplant for Active Crohn's Disease.
Effect of terminal 3'-hydroxymethyl modification of an RNA primer on nonenzymatic primer extension.
On the origin of life.
Genomic analysis of the evolution of fluoroquinolone resistance in Mycobacterium tuberculosis prior to tuberculosis diagnosis.
A highly reactive imidazolium-bridged dinucleotide intermediate in nonenzymatic RNA primer extension.
Characterization of candidate genes in inflammatory bowel disease-associated risk loci.
Proteasome dysfunction triggers activation of SKN-1A/Nrf1 by the aspartic protease DDI-1.
Prevention of Burn-Induced Inflammatory Responses and Muscle Wasting By GTS-21, A Specific Agonist for ?7 Nicotinic Acetylcholine Receptors.
